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The female-specific lethal or sterile effects of mutations in these genes are suppressed by gain-of-function Sxl alleles.

Throughout all but the very earliest period of development, female-specific expression of Sxl is known to be achieved by female-specific splicing of m RNA.

In most wildtype genetic backgrounds, heterozygous females exhibit normal viability and fertility, although occasionally display morphological defects characteristic of early cell death; however, can be dominant semilethal for females in some wild-type genetic backgrounds and under suboptimal growth conditions.

In doubly heterozygous combination with otherwise recessive mutations in positive regulators of Sxl, this allele can behave as a dominant: heterozygote viability is reduced for daughters of da/ females, as well as for females that are also heterozygotes for either sis-a, sis-b or fs(1)A1621.

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Regulates dosage compensation in females by suppressing hyperactivation of X-linked genes.We have recently added many new features, such as private chat, webcam search, sex filter, hd quality video, and more!Free Sex Chat is truly free alternative to My Free Cams, Live Jasmin, i Friends, and Chaturbate!Mutations of Sxl fall into two general classes: (1) recessive loss-of-function alleles that are deleterious to homozygous females, but viable and without phenotypic consequences in males, and (2) dominant gain-of-function alleles that behave as constitutive mutations, dominant and deleterious in males but without adverse effect in females, either heterozygous or homozygous.The variety of functions of the Sxl gene can be affected differentially by mutations, accounting in part for the complex complementation pattern observed for the large array of diverse mutant alleles.

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